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HairGenesis is a safe, natural formulation designed to combat the effects of Androgenic Alopecia, also known as Male Pattern Baldness. The formulation in HairGenesis works by blocking key androgen receptor sites in the hair follicle from damage caused by DHT. Thus the hair follicle is better able to produce a healthier and stronger hair. It is a "natural hairloss pill".

The active ingredients in HairGenesis are derived from naturally occurring nutritional substances, presenting no evidence of negative side effects or drug interactions whatsoever.

The formulation in HairGenesis™, the natural hairloss pill,  works by blocking key androgen receptor sites in the hair follicle from damage caused by DHT. Thus the hair follicle is better able to produce a healthier and stronger hair.

It is a product in oral softgels. Every bottle contains 60 oral softgels.

Here are some components in every softgel: beta sitosterol 50 mg, saw palmetto berry extract (85/95% liposterolic standardized content) 200 mg, lecithin 50 mg, inositol 100 mg, phosphatidyl choline 25 mg, niacin 15 mg, biotin 100 mcg.

1 softgel twice a day.

The product is of high quality.

The HairGenesis™ Revitalizing Oral Softgel ™ is a critical component of the HairGenesis™ system. HairGenesis™ Softgels are designed to be taken orally and work in conjunction with the HairGenesis™ Product Line. Building on the success of the foundational formula - and after years of follow up research - the HairGenesis™ Oral Softgels have recently been dramatically improved and now provide a suite of newly recognized botanical substances that work together to strengthen and protect your hair better than ever before. While results from HairGenesis™ have been reported in as little as two months, a six-month period of use is strongly recommended in order to assess the benefit HairGenesis™ is providing.

This is the rationale behind HairGenesis™ being backed by the most generous customer satisfaction assurance policy in the industry. HairGenesis™ comes with a full 6-month, 100% money-back guarantee. As previously noted, HairGenesis™ Softgels are formulated using naturally occurring nutritional substances, presenting no evidence of negative side effects or drug interactions whatsoever.

HairGenesis™ Oral Softgels - PRODUCT COMPARISON
Shown to inhibit DHT metabolism? NO YES NO YES
Naturally Derived? NO NO NO YES
Orally ingested? NO YES NO YES
Works Systemically? NO YES NO YES
Free from known potential side effects? NO NO YES YES
Suitable for both men and women? YES NO YES YES
Money Back Guaranteed? YES NO NO YES

Other similar products are: Bio-Organics Saw Palmetto Complex, Prostaguard, Proguard and Spencer Forrest Anti-Androgenic Hair Growth Complex.


Phosphatidyl Choline is the main component of Lecithin and is needed for proper mental functioning as well as for efficient breakdown of fats. It is also useful in lowering cholesterol when added to a low fat diet.

Research abstract on Phosphatidyl Choline

Title: Choline availability to the developing rat fetus alters adult hippocampal long-term potentiation.

Jones JP ; H Meck W ; Williams CL ; Wilson WA ; Swartzwelder HS

Department of Genetics, Duke University, Durham, NC, USA.

Brain Res Dev Brain Res, 118(1-2):159-67 1999 Dec 10

Supplementation with choline during pregnancy in rats causes a long-lasting improvement of visuospatial memory of the offspring. To determine if the behavioral effects of choline are related to physiological changes in hippocampus, the effect of perinatal choline supplementation or deficiency on long-term potentiation (LTP) was examined in hippocampal slices of 6-8 and 12-14 month old rats born to dams consuming a control, choline-supplemented, or a choline-free diet during pregnancy. Stimulating and recording electrodes were placed in stratum radiatum of area CA1 to record extracellular population excitatory postsynaptic potentials (pEPSPs). To induce LTP, a theta-like stimulus train was generated. The amplitude of the stimulus pulses was set at either 10% or 50% of the stimulus intensity which had induced the maximal pEPSP slope on the input/output curve. We found that at both ages, a significantly smaller percentage of slices from perinatally choline-deficient rats displayed LTP after 10% stimulus intensity (compared with control and choline-supplemented rats), and a significantly larger percentage of slices from choline-supplemented rats displayed LTP at 50% stimulus intensity (compared with control and choline-deficient rats). Results reveal that alterations in the availability of dietary choline during discrete periods of development lead to changes in hippocampal electrophysiology that last well into adulthood. These changes in LTP threshold may underlie the observed enhancement of visuospatial memory seen after prenatal choline supplementation and point to the importance of choline intake during pregnancy for development of brain and memory function.

Inositol is found as a component of phospholipids in the brain, skeletal, heart, and male reproductive tissues. It functions in nerve transmission, the regulation of enzyme activity and the transportation of fats within the body. Inositol is essential for hair growth and can prevent some cases of thinning hair and baldness. It helps reduce high cholesterol and is important for the heart. Inositol is also beneficial for the nerves, for the treatment of eye abnormalities, eczema and some cases of obesity.

Research abstract on Inositol

Title: Evidence for differential regulation of calcium by outer versus inner hair cells: plasma membrane Ca-ATPase gene expression. Reg

Furuta H ; Luo L ; Hepler K ; Ryan AF

Department of Surgery/Otolaryngology, UCSD School of Medicine, La Jolla, CA 92093-0666, USA.

Hear Res, 123(1-2):10-26 1998 Sep

The expression of mRNA encoding plasma membrane calcium ATPase (PMCA) subunit isoforms (1-4) and splice variants was examined in the adult and developing rat cochlea by PCR and in situ hybridization. High levels of PMCA mRNA expression were observed in the neurons of the spiral ganglion, and in hair cells. Spiral ganglion neurons expressed PMCA 1-3 beginning in embryonic development, reaching high levels shortly after birth, and continuing into adulthood. Inner hair cells expressed PMCA 1 at moderate levels from birth to the time of onset of cochlear function on postnatal day 12, and strongly from then until adulthood. Outer hair cells expressed PMCA 2 at high levels from shortly after birth through adulthood. The data suggest that the calcium clearance requirements of inner and outer hair cells are distinct. PMCA 2 is the isoform with the highest affinity for calmodulin, and has also been associated with high levels of inositol triphosphate. Its presence in outer hair cells suggests that regulation of the enzyme by calmodulin may be particularly important for this hair cell type. It further suggests that inositol phosphate may play a unique role in the outer hair cell.

Research abstract on Inositol

Title: Two new functions of inositol in the eye lens: antioxidation and antiglycation and possible mechanisms.

Ramakrishnan S ; Sulochana KN ; Punitham R

Biochemistry Research Department, Vision Research Foundation, Chennai, India.

Indian J Biochem Biophys, 36(2):129-33 1999 Apr

The extent of glycation of human eye lens proteins with glucose in presence of added inositol was examined in vitro using [U14C] glucose. Lens homogenate was reacted with varying concentrations of glucose and glucose inositol. At the end of the reaction, the proteins were precipitated with TCA, centrifuged, dissolved in NaOH and the radioactivity was measured. Inositol decreased the glycation by 57-67%. Pure inositol and glucose suitably labelled with 3H or 14C when reacted and followed by paper chromatography and HPLC showed that glucosyl inositol was present along with unreacted free glucose. Preliminary studies made of the UV spectra of pure inositol (i) when reacted with H2O2 showed that inositol removed H2O2 from the reaction mixture (ii) when reacted with arachidonic acid showed that they formed a conjugate. The observations indicate that the antioxidant property of inositol could be the result of its' quenching action on reactive oxygen, intermediates and conjugate-formation with compounds like arachidonic acid and the antiglycating property due to scavenging of glucose. The antioxidant and the antiglycating properties of inositol may be beneficial in delaying or averting cataract.

Lecithin is a type of lipid that is required by every living cell in the body. Although lecithin is a lipid, it is partially soluble in water and therefore acts as an emulsifying agent.It has been used as a nutritional supplement for years for the benefit of the liver and maintaining healthy cholesterol.

As an emulsifier, Lecithin enables fats, such as cholesterol and other lipids, to be dispersed in body fluids and properly metabolized. For this reason many people take Lecithin to assist in their weight management.

Research abstract on Lecithin

Title: Diurnal pattern of choline concentrations in serum of pigs as influenced by dietary choline or lecithin intake.

Jakob S ; Mosenthin R ; Huesgen G ; Kinkeldei J ; Poweleit KJ

Hohenheim University, Institute of Animal Nutrition, Stuttgart.

Z Ernahrungswiss, 37(4):353-7 1998 Dec

Athletes especially experience a significant decrease in plasma choline concentrations during exercise which can be compensated in part by consumption of lecithin, a natural source of choline. In addition, the effect of lecithin on plasma choline concentrations in humans is obviously considerably greater and more prolonged than that of an equivalent amount of choline salts. Serum choline acts as a precursor for the synthesis of acetylcholine, which, in turn, acts as a neurotransmitter. The effect of dietary choline derived from either choline chloride or lecithin on the diurnal pattern of free choline concentrations in serum was studied using the pig as a potential model for humans. Six barrows, average initial body weight 120 kg, were fitted with permanent catheters in the jugular vein to determine the diurnal pattern of serum choline concentrations as affected by dietary choline or lecithin intake. The pigs were fed two semi-purified diets twice daily (1,500 g each meal) that contained corn, casein and a mineral-vitamin premix supplemented with equal amounts of choline (480 mg/kg) from either choline chloride or lecithin (BIOFOSFATIN). The diets supplemented with choline were fed at 08.00 h in the morning and the experiment was carried out according to a 3 x 2 cross-over design. All pigs received the basal diet that contained 450 mg/kg choline at the evening feeding (20.00 h). Following an adaptation period of 6 d, blood was collected on d 7; 0.5 h before the morning feeding and 1, 2, 4, 6, 8, 10 and 12 h postprandially. The determination of serum choline concentrations was carried out by tandem-mass spectroscopy. There were no differences (p > 0.05) between the two diurnal patterns of the serum choline concentrations. Both diurnal patterns showed a postprandial peak at 0.5 h (2.71 mg/l for choline chloride and 2.35 mg/l for lecithin supplementation) and decreased after 2 h postprandially to the preprandial concentrations. In conclusion, there were no differences (p > 0.05) in the diurnal patterns of serum choline concentrations in pigs after consumption of dietary choline chloride or lecithin, which is in contrast to corresponding studies in humans.

Niacin is a member of the B complex which aids in promoting a healthy digestive system, is important for the health of the hair and skin, increases circulation and helps reduce blood pressure, lowers cholesterol and triglycerides, promotes relaxation and is necessary for orgasm. High doses may produce a more pronounced niacin flush, which is the tingling, reddening, itching, feeling of warmth produced by the histamine released by niacin. In order to minimize the flush, take niacin with a meal.

Niacin is a member of the B complex. Small doses of niacin are necessary to prevent pellagra. Larger doses can improve histamine release which is necessary for the following: orgasm, lower cholesterol, improve circulation, lower blood pressure, provide a feeling of relaxation and act as a mild growth hormone releaser.

Research abstract on Niacin

Title: Oral niacin prevents photocarcinogenesis and photoimmunosuppression in mice.

Gensler HL ; Williams T ; Huang AC ; Jacobson EL

Arizona Cancer Center, Department of Radiation Oncology, University of Arizona College of Medicine, Tucson 85724, USA.

Nutr Cancer, 34(1):36-41 1999

Topical nicotinamide (niacinamide) has demonstrable preventive activity against photocarcinogenesis in mice. To better understand how this vitamin prevents ultraviolet (UV) carcinogenesis, we tested systemic administration of another form of the vitamin, niacin, and its capacity to elevate cutaneous nicotinamide-adenine dinucleotide (NAD) content as well as to decrease photoimmunosuppression and photocarcinogenesis. BALB/cAnNTacfBR mice were fed the AIN-76A diet supplemented with 0%, 0.1%, 0.5%, or 1.0% niacin throughout the experiment. UV irradiation consisted of five 30-minute exposures per week to banks of six FS40 Westinghouse sunlamps for 22 weeks in the carcinogenesis experiments, yielding a total cumulative dose of approximately 1.41 x 10(6) Jm-2 of UV-B radiation. Dietary supplementation with 0.1%, 0.5%, or 1.0% niacin reduced the control incidence of skin cancer from 68% to 60%, 48%, and 28%, respectively, at 26.5 weeks after the first UV treatment. Two potential mechanisms by which niacin prevents tumor formation were identified. Photoimmunosuppression, critical for photocarcinogenesis, is measured by a passive transfer assay. Syngeneic, antigenic tumor challenges grew to an average of 91.6 19.7, 79.8 11.5, 41.9 11.7, or 13.2 4.1 mm2 in naive recipients of splenocytes from UV-irradiated mice treated with 0%, 0.1%, 0.5%, or 1.0% niacin supplementation, respectively, demonstrating niacin prevention of immunosuppression. Niacin supplementation elevated skin NAD content, which is known to modulate the function of DNA strand scission surveillance proteins p53 and poly(ADP-ribose) polymerase, two proteins critical in cellular responses to UV-induced DNA damage. These results clearly demonstrate a dose-dependent preventive effect of oral niacin on photocarcinogenesis and photoimmunosuppression and establish the capacity of oral niacin to elevate skin NAD levels.

Research abstract on Niacin

Title: Treatment of hyperlipidemia with combined niacin-statin regimens.

Guyton JR ; Capuzzi DM

Department of Medicine, Duke University Medical Center, Durham, North Carolina 27710, USA.

Am J Cardiol, 82(12A):82U-84U; discussion 85-86U 1998 Dec 17

Combined use of niacin with a statin is an attractive option, since these types of medication have the best records in clinical trials for reduction in cardiovascular events and improvement in progression/regression of coronary lesions. In early use, the niacin-statin combination generated a few case reports documenting severe myopathy and rhabdomyolysis. Subsequent prospective trials in >400 patients, however, have not encountered myopathy. This experience includes 165 patients who took a statin in combination with Niaspan, a new, extended-release niacin administered once nightly. Hepatic toxicity with immediate-release niacin and with Niaspan used in combination with statins has been minimal. However, substantial transaminase elevations occurred with the use of a sustained-release niacin (Nicobid) given twice daily. The niacin-statin treatment regimens gave augmented low-density lipoprotein (LDL)-cholesterol reduction along with favorable changes in high-density lipoprotein (HDL) cholesterol, lipoprotein(a), and triglycerides. This combination therapy can be used safely as long as (1) careful attention is given to niacin formulation and dosing; (2) liver functions are monitored; and (3) patients are educated to recognize symptoms of myopathy. However, special caution should apply to use of niacin in combination with high doses of statins, or with statins introduced into clinical practice in 1997 or later, since little experience has accumulated in these circumstances.

Biotin is water soluble and a member of the B complex family. Measured in micrograms (mcg.). Synthesis of Ascorbic acid requires Biotin. The RDA is 150-300 mcg.  Biotin is a coenzyme, essential for the normal metabolism of fats and protein. It maintains healthy skin and is especially good for the health of hair follicles. The best natural sources are nuts, fruits, brewer's yeast and unpolished rice. This vitamin will rejuvenate hair follicles when taken by mouth or rubbed directly on the skin in a cream base. It may even regrow a few hairs, which have gone into early retirement. Hair will stand up straighter and recover a healthy sheen.


DEFICIENCY SYMPTOMS:    Very rare in adults but in infants a condition called seborrheir dermatitis or "cradle cap" (dry, scaly scalp)




Research abstract on Biotin

Title: [Vitamin and dermatology]

Yoshikawa K

Department of Dermatology, Osaka University Graduate School of Medicine.

Nippon Rinsho, 57(10):2385-9 1999 Oct

Vitamin A, B1, B2, B6, B12, biotin, nicotinic acid, panthotenic acid, vitamin C, E and K have been used for various skin disorders. The use is mostly based on the similarity of the skin manifestations seen in their deficiencies, except for the rare cases of clear deficiency like pellagra. Recent introduction of vitamin A and D analogues for psoriasis and keratinization disorders resulted in significant progress in clinical dermatology. Application of vitamin C, E and beta-carotene for UV-induced skin damages are being studied, and the vitamins will be more important in dermatology in the future.

Hair revitalizing shampoo

Hair topical activator serum

What is HairGenesis™?
HairGenesis is the first naturally derived treatment designed to arrest Male Pattern Hairloss. The oral formula is based on a carefully balanced combination of nutritional substances that have been shown to interfere with the hormonal cause of the problem. HairGenesis Activator Serum is a topical formula designed specifically to be used in conjunction with HairGenesis Oral Softgels. This powerful combination works by combating multiple parts of the disorder..

How does HairGenesis™ work?
Male Pattern Hairloss is believed to result from the hormonal conversion of Testosterone to a substance known as DHT (Dihydrotestosterone). DHT binds to specific points on the hair follicle known as androgen receptor sites. This binding causes changes in the morphology (growth pattern) of the hair. The follicle and accompanying structures begin a process of miniaturization. The hair shrinks in diameter. HairGenesis stops this process from occurring by blocking the junction of DHT to the androgen receptor site. In doing so, HairGenesis prevents the cascade of events that result in thinning hair.

Can HairGenesis™ hurt me?
When taken as directed, the answer is no. The substances found in HairGenesis have a long history of clinical use in the treatment of BPH (Benign Prostatic Hyperplasia), another disorder associated with DHT metabolism. These substances have proven themselves very safe.

How long do I have to use HairGenesis before I see results?
It will take three to six months of regular use in order to maximize the potential benefit of HairGenesis. This is due to the slow rate at which your hair grows. (average 1/2" per month)

What is the key ingredient in HairGenesis?
The formulation in HairGenesis contains precise amounts of specific ingredients noted on the label. All ingredients are necessary in order to achieve the excellent results found with the use of HairGenesis. Just as a gourmet recipe requires an exact blending of specific ingredients at precise levels, so too HairGenesis is an exquisitely precise combination of nutritional substances designed to protect your hair.

Why do I need to continue using HairGenesis even after I start to see results?
Your body is continuing to produce DHT. DHT causes hairloss. HairGenesis fights DHT.

Will HairGenesis cause unwanted hairgrowth in areas other than my scalp?
Absolutely not. In fact the same metabolic mechanism that causes Male Pattern Hairloss is believed to influence secondary hairgrowth in other areas of the body. Therefore, HairGenesis Oral SoftGels may actually help reduce the onset of (DHT mediated) unwanted facial and body hair.

What about drug interactions while taking HairGenesis?
HairGenesis, taken as directed, is free from known drug interactions.

When should a person begin taking HairGenesis?
HairGenesis is recommended for individuals over age 18 who have begun experiencing the symptoms of Male Pattern Hairloss.

Does HairGenesis rejuvenate dead hair follicles?
No. HairGenesis is not a miracle cure. However, Male Pattern Hairloss presents primarily as a qualitative change. Meaning that each effected hair becomes progressively thinner over time. HairGenesis works by allowing the hair follicle (the factory that produces the hair) to produce a healthier product. The theory being if you have ten hairs that are twice as thick, it is as good as having twenty hairs.

Does HairGenesis also work for women?
Yes. Because DHT also causes Female Pattern Hairloss, currently affecting over 20 million American women. HairGenesis has been shown to work very well in this population, thus representing an excellent therapy option for women interested in combating this disorder.

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